Selank (10mg)
Rated 4.8 out of 5
$89.98
A synthetic analog of the endogenous tetrapeptide tuftsin (Thr-Lys-Pro-Arg), Selank is a neuropeptide studied for its influence on neurological and behavioral pathways. Research highlights its anxiolytic and nootropic effects, alongside immunomodulatory and anti-inflammatory properties. Requires reconstitution before use.
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Research Use Only: All materials are supplied exclusively for in vitro research and laboratory work by qualified professionals and are not marketed as drugs, supplements, or medical products.
A synthetic analog of the endogenous tetrapeptide tuftsin (Thr-Lys-Pro-Arg), Selank is a neuropeptide studied for its influence on neurological and behavioral pathways. Research highlights its anxiolytic and nootropic effects, alongside immunomodulatory and anti-inflammatory properties. Requires reconstitution before use.
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This material is sold strictly for qualified laboratory and in vitro research. It is not a drug, food, dietary supplement, or cosmetic and is not approved for human or animal use. Any attempt to ingest, inject, or otherwise introduce this material into the body is prohibited. Misuse, misbranding, or reselling this material for non-research purposes is strictly forbidden. Information provided on this site is for educational purposes only. The statements made within this website have not been evaluated by the U.S. Food and Drug Administration (FDA). Neither the statements nor the products offered by this company are intended to diagnose, treat, cure, or prevent any disease.
Table of Contents
1. Characteristics
2. How is Selank Used in Research?
Selank engages several neurobiological pathways simultaneously, distinguishing it from more narrowly targeted compounds. Documented interactions include modulation of BDNF and its TrkB receptor, which govern neuronal survival and synaptic plasticity, alongside activity within the GABAergic system that underlies its anxiolytic profile. Monoaminergic regulation, specifically serotonin and dopamine pathways tied to mood and cognition, represents an additional layer of its mechanism. Beyond neurobiology, cytokine modulation, including IL-6 and TNF-α activity, natural killer cell enhancement, and antioxidant protection against oxidative stress, reflects its immunomodulatory reach.
3. Areas of Study
- Stress Pathway Activity: Experimental models have examined Selank's influence on stress-related signaling, with particular focus on inhibitory neurotransmission and behavioral regulation under stress conditions.
- Mood Modulation: Research has investigated its role in monoaminergic function and BDNF expression as they relate to mood-associated neural circuitry.
- Cognitive Processing: Spatial and verbal task performance, attention regulation, and information processing mechanisms have all been explored in relation to Selank's activity in cognitive pathways.
- Neuronal Function: Interactions with BDNF expression and oxidative stress responses have been studied in the context of cellular resilience and neuronal integrity under stress-related conditions.
- Immune Signaling: Cytokine activity, cellular immune responses, and broader inflammatory signaling pathways remain active areas of investigation in experimental settings.
4. Summary
Across neurobiological and immunological research, Selank occupies a position of sustained scientific interest due to the breadth of systems it appears to engage. Current findings illuminate its activity across stress, mood, cognition, and immune signaling, though comprehensive mechanistic characterization across diverse experimental models remains an ongoing pursuit.
5. References
- 1. Konstantinopolsky MA et al. Selank, a Peptide Analog of Tuftsin, Attenuates Aversive Signs of Morphine Withdrawal in Rats. Bulletin of Experimental Biology and Medicine, 2022. PubMed 36322304
- 2. Volkova A et al. Selank Administration Affects the Expression of Some Genes Involved in GABAergic Neurotransmission. Frontiers in Pharmacology, 2016. PubMed 26924987
- 3. Kolik LG et al. Selank, Peptide Analogue of Tuftsin, Protects Against Ethanol-Induced Memory Impairment by Regulating BDNF Content in the Hippocampus and Prefrontal Cortex in Rats. Bulletin of Experimental Biology and Medicine, 2019. Source
- 4. Filatova E et al. GABA, Selank, and Olanzapine Affect the Expression of Genes Involved in GABAergic Neurotransmission in IMR-32 Cells. Frontiers in Pharmacology, 2017. PubMed 28293190
6. Research use
All products on this site are intended exclusively for research and development use. Products are not for human or animal consumption of any kind.
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